The Significance of Hydrophilic Linkers in the Development of ADC

2024-07-17 00:53:54
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With the development of ADCs, hydrophilicity must be considered. Low hydrophilicity of the linker shows the following disadvantages[1-4]:

(1) low conjugate efficiency and DAR

(2) polymerization and sedimentation in human plasma

(3) off-target toxicity by nonspecific uptake

(4) undesirable pharmacokinetics by rapid elimination from plasma


Simmons et al. [5-8] studied MMAE-based ADCs with linkers containing 0, 4, 8, or 12 PEG units to investigate the relationship between off-target toxicity and hydrophilicity. In their tolerance experiments, all mice in the PEG0 ADC group died by the 5th day at a 20 mg/kg dose, while the PEG8 and PEG12 groups had a 100% survival rate after 28 days. Immunohistochemical (IHC) staining of mouse livers showed that PEG0-containing ADCs had nonspecific uptake and released a large quantity of MMAE after 2 hours at 10 mg/kg. It was therefore proven that the low dose tolerance of ADCs with low hydrophilicity was caused by nonspecific liver absorption. ADCs with PEG12 had slower plasma clearance (7.3 mL·kg/day) and longer plasma exposure than PEG0 ADCs (>46.3 mL·kg/day). Thus, ADCs with higher hydrophilicity improve pharmacokinetic parameters and reduce nonspecific uptake and off-target toxicity.


Incorporating PEG, sulfonate, phosphate, and other hydrophilic groups into linkers is a popular way to enhance ADC’s hydrophilicity. Currently, incorporating PEG into ADC linkers is very common practice. For instance, marketed drugs like Trodelvy® and Zynlonta® incorporate PEG.


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Reference

[1] Acta Pharmaceutica Sinica B, 2021, 12, 3889-3907

[2] Drug Metab Dispos, 2021, 42, 1914–1920

[3] Nat Biotechnol, 2015, 33, 733–735

[4] Bioconjug Chem, 2017, 28, 1371–1381

[5] Pharmacol Ther, 2018, 181, 126-142

[6] Blood, 2018, 10, 1094-1105

[7] Hebei Medicine, 2019, 37, 18-22

[8] Toxicol Appl Pharmacol, 2020, 392, 114932